RichGotsPots

Literature Summary and Critique Iwamoto M, et al. Klinische Wirkiung von Crataegutt bei Herzerkrankungen ischasemischer und/oder hypertensiver Genese. Planta Med 1981;42:1-16. A hawthorn preparation containing 30 mg hawthorn extract, standardized to 1 mg procyanidins, was used in a double-blind controlled study of 80 patients (35 active, 45 placebo). The hawthorn group reported a greater overall improvement of cardiac function, dyspnea, and palpitations. Improvements in ECGs were not found to differ. Nausea, fatigue, and sweating were reported in the hawthorn group. Schmidt U, et al. Efficacy of the hawthorn preparation in 78 patients with chronic congestive heart failure defined as NYHA functional class II. Phytomedicine 1994;1:17-24. Seventy-eight patients were administered hawthorn extract 600 mg/day or a placebo. After a one-week washout, patients were treated for eight-weeks. An ergometer bicycle was used to determine working capacity at baseline and during treatment. Between day zero and fifty-six, the working capacity of those taking hawthorn increased by 28 watts compared to 5 watts in those receiving placebo. There was a significant decrease in systolic blood pressure and heart rate as compared to placebo. Top Dosage Oral: Many different formulations exist; dosages vary between products. NYHA stage II cardiac insufficiency: 160-900 mg extract (standardized 4-20 mg flavonoids / 30-160 mg oligomeric procyanidins) daily in divided doses for 6 weeks or 120-240 mg extract (standardized to 1.8% vitexin rhamnoside/10% procyanidins) three times a day for 6 weeks.

Mechanism of Action It is thought that hawthorn causes direct dilation of smooth muscle in coronary vessels thereby lowering their resistance and increasing blood flow. Hawthorn is also characterized as having positive inotropic effects leading to an increase in heart rate, nerve conductivity, and heart muscle irritability (4). Hmmmm not sure if this will lead to more blood pooling and more tachy

Derived from the flower, leaves, and fruits of the plant. Hawthorn has been used as an digestive aid in traditional Chinese medicine for centuries. But currently, it is used primarily to treat heart failure. Hawthorn extract exhibited anti-inflammatory, gastroprotective, and antimicrobial properties in vitro (15). Clinical studies suggest effectiveness against congestive heart failure (10) (12) (14) and diabetes (13). Further research is needed to establish efficacy. Frequently reported adverse events include nausea, sweating, and fatigue. Signs of overdose include hypotension and arrhythmias.

Side effects Overdose can cause cardiac arrhythmia and dangerously lower blood pressure. Milder side effects include nausea and sedation.[16]

Has anyone tried it? The claims seem remarkable especially for our condition. Several pilot studies have assessed the ability of hawthorn to help improve exercise tolerance in people with NYHA class II cardiac insufficiency compared to placebo. One experiment, at (300 mg/day) for 4 to 8 weeks, found no difference from placebo. The second study, including 78 subjects (600 mg/day) for 8 weeks, found "significant improvement in exercise tolerance" and lower blood pressure and heart rate during exercise. The third, including 32 subjects (900 mg/day) for 8 weeks, found improved exercise tolerance as well as a reduction in the "incidence and severity of symptoms such as dyspnea" and fatigue decreased by approximately 50%.[6] In the HERB-CHF (Hawthorn Extract Randomized Blinded Chronic HF Study) clinical study, 120 patients took 450 mg of hawthorn extract twice daily for 6 months in combination with standard therapy and a standardized exercise program. "No effects of hawthorn were seen on either quality-of-life endpoint (Tables 1 and 2), or when adjusted for LVEF".[14] One research program, consisting of 1,011 patients taking one tablet (standardized to 84.3 mg procyanidin) twice daily for 24 weeks, found "improvements in clinical symptoms (such as fatigue, palpitations, and exercise dyspnea), performance and exercise tolerance test, and ejection fraction".[15]

I came a across two interestive IV rehydration. Has anyone had either. Ringer's lactate solution- its mainly used for diarrhea Hydroxyethyl starch- its mainly used for Hypovolemic shock, which made me think it would be useful

Yup this has happened to me more times than I can count.. Rheumy wouldn't test anything. Neuro muscular doc would do any testing.. Pulm wouldn't do any testing. I just keep pushing on. Occasionally u find a doc that is open minded and will go above the call of duty, but those are rare. I've had 3-4 though, so don't give up..

Lidocaine is the most popular Caine these days but there are tons more. There are brands like carbocaine that are the same as lidocaine and can come with epi but also more often don't for people who can't tolerate epi... Has anyone been to a natural holistic type of dentist? I wonder what they use?

Did any of you ever read the full Gregg Page story? Well he thought he had OI for around 15 years but about a year after his hernia surgery it got way worse. Similiar thing happened to me. I had Undiagnosed POTS in 2006 then in 2009 I had nasal polyp surgery and the. About one year later pots started to creep back until it got unbearable and has been unbearable for the last year almost... Would be curious to hear other stories..

That is so awesome! I wish you all the luck and never to get another flare ever!

Looks like a good responses so far. I purposely didn't put mito, because I've heard so many people say they suspect it. I also don't feel it's related to BP issues as much as the other choices are... But if it was confirmed that I thought it would be interesting..

Just curious because I have both of these myself, which I think make me much harder to treat..

Nmpotsie, I had the worst reaction to percocets, yucky for me... But brave of you, maybe look into those laser dental treatments in the future.. Btw in my research I saw that if you have an allergy to one anesthetic it doesn't mean u r allergic to them all..

I can tell you that I'm not hyper mobile or eds, if anything I'm the opposite lol...

Mcblonde my co2 is usually normal too but and they even monitored it when I was having breathing issues.. Hmmm. But Dr. Stewart has observed it, so maybe it's happening in or blood rather than or breath.. Did they do a blood/gas atrial test? It hurts so they usually dont, like to puncture an artery.. But that the only true way to find out. They do that for COPD patients because they get co2 trapped in their lungs and it doesn't show up on end tidal breath tests...

Sue I'm with you maybe I'll post about hyper and pooling..

Gloria, in my initial post I listed what are potential causes for either one.

Alan, why not do florinef and the BP at the same time if u think it will help?

Looks like a lot of people voted so far and it's not that bias, split down the middle. I do think there is correlation here that u would get in a dentist office because they don't know at pots and you would get it in a clinic per se because they don't do dental work lol

Dr Stewart says 50% of us have Hyperpnea, it's a form of hyperventilation that isn't rapid it's deep breathing and it cause co2 levels to drop. So I've done some research on Hyperpnea. Hyperventilation syndrome is similar and the way they treat is with breathing exercise 2-3x a day for 10-15min a day. It helps to retrain the ans to breath properly. I also found a study that of meds that may treat it http://www.jci.org/articles/view/116569. The only problem is they are only in clinical trials currently, but they also seem to point to some meds like zyflow that may help... Might be indirectly similiar to a mast cell issue, not straight up MCAD..

This happens to me all the time!

BTW this is part 1 http://forums.dinet.org/index.php?/topic/20049-dysautonomia-breathing-related-issues-and-treatments/

reposting to try to get more vote

I just read on a another forum that a few POTS people, need to get a ton of injection before they are numb? I seriously think I got like 10+ injection one time and still felt it.. Just wanted to know if it was coincidence or that these other pots people have the same thing.. P.S. I know most dentists don't use Novacaine anymore, but that's what people usually associate with local anesthesia :-) BTW I found that these patients usually need a lot of Novacaine too http://en.wikipedia.org/wiki/Hypokalemic_sensory_overstimulation

posting to try to get more poll takers