ramakentesh Posted February 21, 2013 Report Share Posted February 21, 2013 Well worth a read:http://www.neurology-asia.org/articles/neuroasia-2012-17(2)-155.pdfhttp://www.neurology-asia.org/articles/neuroasia-2012-17(2)-155.pdf Quote Link to comment Share on other sites More sharing options...
HopeSprings Posted February 21, 2013 Report Share Posted February 21, 2013 Interesting! A bit difficult for me to understand. I would be interested in hearing how others interpret their findings. I also have questions if anyone can answer them:Low thoracic fluid content (18 L/k Ohm), reflecting the low totally fluid volume in the chest cavity related to postural tachycardia, was observed by transthoracic electrical bioimpedance method. So there is a way to look at this?In contrast to the nerve conduction studies of the lower extremity which showed no peripheral sensorimotor neuropathy,the prolongation of sympathetic skin response(SSR) latency (2450 milliseconds) in legs demonstrated peripheral autonomic denervation. What is this? QSART?In conclusion, we propose that peripheral autonomic denervation of legs from thoracic spinal cord atrophy and demyelination in our patient may be the pathophysiology of her POTS. I don't understand. Sounds like they're saying the cord problem is directly responsible for the neuropathy in the legs? Quote Link to comment Share on other sites More sharing options...
ramakentesh Posted February 21, 2013 Author Report Share Posted February 21, 2013 Apparently they can test thoratic volume, that test is different to qsart and an interesting take on per ventricular are of brain and spine may relate to leg denervation. I just saw one of the major docs is on twitter - maybe I'll tweet him this? Quote Link to comment Share on other sites More sharing options...
ramakentesh Posted February 21, 2013 Author Report Share Posted February 21, 2013 Sorry peri ventricular and area! Quote Link to comment Share on other sites More sharing options...
HopeSprings Posted February 21, 2013 Report Share Posted February 21, 2013 Yes, tweet away! Quote Link to comment Share on other sites More sharing options...
looneymom Posted February 21, 2013 Report Share Posted February 21, 2013 Very interesting article. Keep them coming. Quote Link to comment Share on other sites More sharing options...
sue1234 Posted February 22, 2013 Report Share Posted February 22, 2013 Just as I had thought on some of my previous MS posts, I think some of the etiology for MS will eventually open a door to some of the causes for some our our POTS. We have too many "jump out at you" things that are common with so many...so many have neck issues or spine/posture issues. Don't know the exact mechanisms, but something may show up from that area.I think mechanical issues are just as strong as a suspicious factor as antibodies. Definitely let us know if you are able to grab any more information. Quote Link to comment Share on other sites More sharing options...
jangle Posted February 22, 2013 Report Share Posted February 22, 2013 Neuronal loss is unlikely cause given that POTS can begin and end spontaneously. Links to MS probably more akin to inflammation or possible autoantibody involvement. Quote Link to comment Share on other sites More sharing options...
ramakentesh Posted February 22, 2013 Author Report Share Posted February 22, 2013 Yeah very true. I was wondering whether longterm reductions in cerebral blood flow might mess up the periventricular area of the brain and result in reduced blood volume. Quote Link to comment Share on other sites More sharing options...
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