Jump to content
Search In
  • More options...
Find results that contain...
Find results in...

haugr

Members
  • Content Count

    163
  • Joined

  • Last visited

Community Reputation

0 Neutral

About haugr

  • Rank
    Advanced Member

Profile Information

  • Gender
    Male

Recent Profile Visitors

The recent visitors block is disabled and is not being shown to other users.

  1. Zoloft has been a positive for me.
  2. I get that too. I noticed with me that my ears got bright red as I was coming down from an hyperadrenergic episode. I have a theory that it happens after severe vasoconstriction as the blood vessels dilate and blood flow increases. I have seen a couple of papers suggesting Red Ear Syndrome is the same as Erythromelalgia of the ears.
  3. I think that there is a good chance that those of us with with POTS caused by an ACE2 deficiency are coronavirus resistant since coronavirus infects cells by binding to the very thing that is broken in us. The outlook might even be better for those that are taking Losartan to treat their ACE2 deficiency since that is a med that is currently being used in clinical trials as a coronavirus treatment. Having said that though, I am not taking any chances. I am staying as quarantined as possible until a vaccine is out.
  4. Where did you have those tests done?
  5. I am not really sure. Julien Stewart had reported some positive results from increasing anti-oxidants though.
  6. I wouldn't. Too many non-gaurantees for such a permanent decision IMHO.
  7. No kidding. I wish these tests were commonly available.
  8. I found that paper about high angiotensin II levels in some potsies and its effects on renin: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4511483 A subset of POTS patients with increased vasoconstriction related to decreased bioavailable NO (nitric oxide) have decreased blood volume Ang II reduces bioavailable Nitric Oxide Ang II can increase the release of noradrenaline (norepinephrine) from ganglionic and postganglionic sympathetic nerves Ang II promotes oxidative stress by causing the formation of superoxide radical through its effects on NADPH oxidase. The interaction with superoxide reduces the bioavailable Nitric Oxide Ang II is part of a control loop with feedback inhibition of renin production. Thus increased exogenous or endogenous plasma concentrations of Ang II suppress the release and expression of renin
  9. It makes sense to me that clonidine would have those negative effects since clonidine primarily suppresses norepinephrine instead of epinephrine. An alpha-beta blocker would probably help get the blood pressure down. Might want to also get tested for a pheochromocytoma if it hasn't already been done.
  10. I've never heard of infections being associated with pots. The others I think could possibly be attributed to high adrenaline. I would recommend looking up the symptoms of a pheochromocytoma since it will give a good description of what happens when your adrenaline gets out of control.
  11. I think that mine is genetic. I have 4 biological siblings and 2 of them, while not officially diagnosed, are convinced that they also have it. My sister had been dealing with what she thought were panic attacks her entire life until she did the poor mans tilt table after I was diagnoses and she turned out to meet the criteria. Since then she has been pounding the sports drinks which has helped her immensely. My brother has really high blood pressure and low testosterone that he was diagnosed with in his late 20s. He also is on a high salt diet and lisonopril (which reduces Angiotensin II). I have another cousin in her early 20s that was passing out from time to time until she was put on a high salt diet. I should probably submit myself and my family to a genetic researcher
  12. That actually sounds like the perfect summer to me
  13. What causes this particular form of hyper pots (or Low Flow POTS as Julien Stewart calls it), is that there is too much Angiotensin II in the blood stream because the ACE2 is broken and fails to catabolize the Angiotensin II properly into the lesser forms of Angiotensin known as Angiotensin 1-7. The kidneys then have a feedback loop that suppresses the release Renin when Angiotensin II levels are high. So... why does salt help? Well, the main goal is to get your Angiotensin II levels down to a tolerable level. Salt and extra fluids raises your blood volume which leads to a state where your RAAS doesn't even really need to activate which allows your Angiotensin II levels to finally decrease as it gets slowly gets converted to Aldosterone which actually ends up lowering your blood pressure. The raised blood volume also gets the other POTS symptoms under control. Julien Stewart also recommends Losartan as a way to suppress the effects of the high Angiotensin II levels.
  14. Salt with hyperpots is tricky. You pretty much have to salt supplement if your symptoms are due to hypovolemia, but if you take too much though it will cause your bp to go higher, from what I understand. In your case, I would guess the frequent urination is a pretty goid sign that you are likely hypovolemic.
  15. @Outaker I am actually so glad to hear that you were able to get confirmation. I know it has been a long and drawn out battle for you. Were you able to find a local clinic to do the standing vs suppine catechlomines test?
×
×
  • Create New...