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POTLUCK

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About POTLUCK

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  1. Will their be any published results from the study done July 2014 with a larger number of people tested than the original 14/14 study. Also, if not published yet or if this will not be published does anyone know anything about the actual results? How many people in the larger study had the antibodies? Did it show up in different types of patients such as Hyperadrenergic? It is good news that they have obtained an even larger grant to work on this but it has been 8 months, shouldn't results be available from the July 2014 study?
  2. Did anyone hear anything on the larger study?
  3. I hear they are doing the follow up study in early July at a conference, so maybe there will be more info. available soon. This topic is very interesting, and may prove to be the cause of POTS in most patients.
  4. The comments about this study on the Dysatonomia international website seem to indicate they are certain the POTS patients had antibodies and the controls did not. As I said the paper itself did not explain potentials for error on this. Maybe they are certain the POTS patients all had antibodies, but if that is so it would seem most of diagnosed POTS patients will have them, as it is too coincidental for 14/14 at two centers.
  5. Thank you for the replies. Chaos- Do you know what antibodies they were testing for at Mayo in regard to the 14%, and is this a test that is available to a patient who wants it? The JAHA study is very hard to understand both medically and statistically. I have a little insight medically ( I.e. B1 & B2 receptor antibodies acting as both agonists and antagonists and also thus causing reflex actions such as an increase in the persons own Epi/NE response) This makes a lot of sense and could account for many "odd" things in POTS ( such as my more Hyperadrenergic POTS picture, or people's diffe
  6. It seemed like a bad idea to steal the other thread on this. My computer will not let me post a link to the article so if someone else can that would be great. " Autoimmune basis for Postural Orthostatic Tachycardia Syndrome" The questions are: 1. It appears all 14 POTS patients and none of the controls had autoantibodies to Adrenergic Receptors AND varying effects. (agonist/antagonist, B1, B2 etc.) A. How sure are they that the POTS patients had autoantibodies, and the controls did not. (i.e. It seems common sense wise that if ALL POTS and no controls have something it is not chance,
  7. Still not back, no idea what it was or why but gone for now.
  8. This strange and worry some symptom that went on for some time in a fluctuating pattern went away at the beginning of September and never came back.
  9. This has not been preset for the last week. I am hoping it stays this way-gone.
  10. Another month gone by and this earlier statement still applies. "This comes and goes but has not gotten better, and is still in the same spot and I still do not know what it is."
  11. This comes and goes but has not gotten better, and is still in the same spot and I still do not know what it is.
  12. The beet root juice had a very clear effect on me, with a very uncomfortable rebound. You might find this stronger with a more clear answer to if it is doing something for those not seeing an effect. It is available bottled.
  13. Wonderful news. I have been doing well with my POTS, but only when I keep running, as soon as I stop, the standing HR numbers rise drastically. I do believe cabbage is one of the foods like Beet root juice that are high in nitrates and thus effects nitric oxide which dilates the blood vessels. In the past I found countless articles on this, but with the beet root juice I got a sharp rebound effect the same day. I also can't really do juices well with Diabetes. There is information on nitric oxide and POTS, CFS, Temporal Lobe Epilepsy, reductions due to Diabetes, relation to Melatonin, and ant
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